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lack of NADH-coenzyme Q reductase相关文献:
Ndufs4 knockout mouse models of Leigh syndrome: pathophysiology and intervention.
van de Wal MAE, Adjobo-Hermans MJW, Keijer J, Schirris TJJ, Homberg JR, Wieckowski MR, Grefte S, van Schothorst EM, van Karnebeek C, Quintana A, Koopman WJH.
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Global mitochondrial protein import proteomics reveal distinct regulation by translation and translocation machinery.
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Mitochondrial ubiquinol oxidation is necessary for tumour growth.
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Cellular mechanisms of complex I-associated pathology.
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Mammalian Mitochondrial Complex I Structure and Disease-Causing Mutations.
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Trends Cell Biol. 2018 Oct;28(10):835-867. doi: 10.1016/j.tcb.2018.06.006. Epub 2018 Jul 26.
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Saturation kinetics of coenzyme Q in NADH oxidation: rate enhancement by incorporation of excess quinone.
Fato R, Bernardo SD, Estornell E, Parentic Castelli G, Lenaz G.
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On the Edge of Dispensability, the Chloroplast ndh Genes.
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Cryo-EM structures of Na+-pumping NADH-ubiquinone oxidoreductase from Vibrio cholerae.
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Cyclophilin D, Somehow a Master Regulator of Mitochondrial Function.
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